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www.ncbi.nlm.nih.gov/pubmed/26339034

Science. 2015 Sep 4;349(6252):aac6284. doi: 10.1126/science.aac6284. Epub 2015 Sep 3. The predator-prey power law: Biomass scaling across terrestrial and aquatic biomes. Hatton IA(1), McCann KS(2), Fryxell JM(2), Davies TJ(3), Smerlak M(4), Sinclair AR(5), Loreau M(6). Author information: (1)Department of Biology ...

www.ncbi.nlm.nih.gov/pmc/articles/PMC4223253

Nov 7, 2014 ... Gukovsky et al[121] used PI3Kγ-deficient mice as well as pharmacologic PI3K inhibitors to investigate the role of PI3K in CCK-induced responses in isolated pancreatic acinar cells. .... Saluja AK, Lerch MM, Phillips PA, Dudeja V. Why does pancreatic overstimulation cause pancreatitis? Annu Rev Physiol.

academic.oup.com/edrv/article-pdf/23/3/303/8860400/edrv0303.pdf

V. Identification of the Susceptibility Genes. VI. Animal Studies. VII. ..... The most common source of trauma in the elderly is falls. Most age-related osteoporotic fractures, particularly at the. Peacock et al. • Genetics of Osteoporosis. Endocrine ...... Kaste SC, Chesney RW, Hudson MM, Lustig RH, Rose SR, Car- bone LD 1999 ...

onlinelibrary.wiley.com/doi/pdf/10.1113/jphysiol.2009.172254

Large, 2002; Flemming et al. 2002; Wilson et al. 2002;. Weirich et al. 2005; McElroy et al. 2008; Ng et al. 2008). CCE is activated in response to Ca2+ release induced by agonists activating receptors coupled to the inositol 1,4,5- trisphosphate (IP3) signalling pathway, or by agents that inhibit the SR Ca2+- ATPase (SERCA), ...

onlinelibrary.wiley.com/doi/full/10.1046/j.1365-2141.2003.04726.x

Dec 11, 2003 ... Linkage analysis in four HLH families of Pakistani origin revealed a putative disease gene on chromosome 9q21.3–22 (Ohadi et al, 1999). Another region on chromosome 10q24 was then identified in linkage with the disease. The locus encodes for perforin, a protein that mediates the cytotoxic activity of ...

ftp://ftp.cdc.gov/pub/publications/mmwr/rr/rr58e324.pdf

Mar 24, 2009 ... Soriano V, Dona C, Rodriguez-Rosado R, Barreiro P, Gonzalez-Lahoz J. Discontinuation of secondary prophylaxis for opportunistic infections in HIV- infected patients receiving highly active antiretroviral therapy. AIDS 2000;14:383 –6. 169. Zellweger C, Opravil M, Bernasconi E, et al. Long-term safety of dis-.

journals.sagepub.com/doi/full/10.1086/680189

Arreola et al. were the first to propose a kinetic model describing the Ca2+ and voltage dependence of CaCCs recorded in parotid acinar cells: ...... Ca2+- induced Ca2+ release (CICR), which can provide an additional stimulus for ANO1 activation by CaV1.2; (8) spontaneous Ca2+ release by RyR in the SR, giving rise to ...

www.physiology.org/doi/full/10.1152/ajpcell.00341.2015

Sep 12, 2016 ... CaMKII regulates Ca2+ influx in cardiomyocytes by modulating Cav1.2 channels and the associated Ca2+ current (16, 194). Furthermore, McCarron et al. (103) showed that inhibition of CaMKII activity prevented the increase in Cav1.2 current elicited by Ca2+ in smooth muscle cells. In addition to Ca2+ ...

www.frontiersin.org/articles/10.3389/fmicb.2016.00467/full

Different sandfly species from the genera Phlebotomus or Lutzomyia transmit the parasite to human. Factors such as the parasite number, species and site of invasion, sandfly saliva, host-derived factors affecting immune-competency and the host–parasite interaction determine the severity of disease (Rodrigues et al.,  ...